Cardiac Hypertrophy and Failure
Libbey, 1990 - Heart - 696 pages
Cardiac insufficiency, a major cause of premature mortality, is a key focus of medical and pharmaceutical research. This book aims to bring clinicians and researchers up-to-date on recent biophysical, cellular physiological and molecular biological developments and their clinical applications.
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Experimental models P Y HATT
The origins of cardiac hypertrophy B SWYNGHEDAUW
Cardiac metabolism and energy exchange A YOUNES
39 other sections not shown
acid actin activity adaptation adrenergic adult afterload alterations angiotensin aortic insufficiency aortic stenosis arrhythmias arterial hypertension ATPase atrial natriuretic Biochem Biol Chem Ca2+ calcium cardiac failure cardiac hypertrophy cardiac insufficiency cardiac muscle cardiomyopathy cellular cent changes chronic Circ Res Circulation clinical collagen congestive heart failure contractile proteins contraction coronary decrease diastolic dilated disease ejection fraction end-systolic experimental expression factor fibres fibrosis filaments gene haemodynamic heavy-chains hormone hyperthyroidism hypertrophied hearts hypertrophied myocardium increase induced infarction inotropic intracellular ischaemia ischaemic isoforms isolated isomyosin left ventricle left ventricular hypertrophy light-chain load mechanical overload membrane metabolism mitochondria Mol Cell Cardiol mRNA myocardial hypertrophy myocardium myocytes myosin heavy chain Na+/K+-ATPase normal patients perfusion Physiol pressure overload protein synthesis rabbit rat heart receptors regulation relaxation response sarcomere sarcoplasmic reticulum skeletal muscle spontaneously hypertensive rats stimulation studies suppl systolic tissue tropomyosin troponin vasodilators ventricular function volume overload wall stress