CRC Desk Reference for Allergy and Asthma
CRC Press, Apr 26, 2000 - Medical - 400 pages
As the prevalence of allergic diseases and asthma has increased in recent years, scientists have intensified research efforts to understand the pathogenesis of these sometimes debilitating conditions. Indeed, researchers have provided key insights-and new concepts-that have changed therapeutic strategies. Moreover, allergic inflammation has served as something of a model in furthering our understanding of regulatory mechanisms of inflammation in general.
Presented in an encyclopedic format, the CRC Desk Reference for Allergy and Asthma provides the latest scientific information on allergic diseases and asthma. It combines knowledge from clinical immunology with basic immunology, pharmacology, biochemistry, and molecular biology to examine new treatment strategies for allergic diseases and asthma.
Highlights of the CRC Desk Reference for Allergy and Asthma include:
Written by leading authorities in the field, the CRC Desk Reference for Allergy and Asthma serves as a timely reference for anyone interested in this area of research. Not only does it present information in an easy-to-use format, but it addresses many hot topics and views that will have sustaining value for years to come.
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activated T cells adhesion molecules airway hyperresponsiveness airway obstruction allergen allergic diseases allergic inflammation antibodies antigen antigen-presenting cells antigen-presenting cells APCs apoptosis associated atopic dermatitis basophils binding caspases cause CD4+ T cells CD8+ cell activation cell antigen receptor cell surface chemokines chronic clinical cloning co-stimulation Coombs and Gell corticosteroids cytokine gene cytokine receptors delayed hypersensitivity dendritic cells differentiation dose drugs effects endothelial cells enzyme eosinophils epitopes exposure expression function Gell classification genetic GM-CSF granules granulocytes high-affinity IgE receptor histamine house-dust mite IFN-y IgE antibodies immune response immunoglobulin immunosuppressive immunotherapy increased induce infection inflammatory cells inhaled inhibition inhibitors interactions intracellular leukocytes leukotrienes ligand lung lymphocytes macrophages mast cells mechanisms mediators MHC class monocytes neutrophils NK cells pathways peptide pollen production proteases proteins release respiratory rhinitis role sensitization sequence signal transduction smooth muscle specific stimulation surface molecules symptoms syndrome Th2 cells Th2 cytokines therapy tissue transcription factors urticaria