Practical Pediatric HematologyAnupam Sachdeva Evidence-based medicine is the need of the hour. Every pediatrician strives for perfection is his or her practice. These are exciting times for Pediatric Hematology and Oncology. Major advances during the past decade in the field have enhanced the understanding and significantly influenced the management and outcome of many of these chronic and fatal diseases affecting children. While opportunities to learn decrease after one leaves the medical school, science keeps evolving. Keeping this in mind, Indian Academy of Pediatrics (IAP) envisaged starting specialty training under its plan of action 2006. Such trainings keep professional and residents updated about newly launched technologies and other advancements. This book is well illustrated and has many pictures and diagrams for the quick understanding of readers. This will be of use not only to the postgraduates of pediatrics but also will be of immense use to a general pediatrician as a ready-reckoner to be kept on the desk. |
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Page 2
... abnormalities. In patients with severe hemolysis: • Nucleated erythrocytes (sometimes accompanied by granulocytosis and thrombocytosis) • Schistocytes in immune mediated hemolytic anemia and hereditary spherocytosis • Spiculated ...
... abnormalities. In patients with severe hemolysis: • Nucleated erythrocytes (sometimes accompanied by granulocytosis and thrombocytosis) • Schistocytes in immune mediated hemolytic anemia and hereditary spherocytosis • Spiculated ...
Page 3
... Abnormalities Dohle bodies: They are bluish cytoplasmic inclusions that can be seen in the neutrophils from patients with: • Bacterial infection • Burns • Myelodysplasia • May-Hegglin anomaly • Pregnancy. Alder-Reilly bodies: They are ...
... Abnormalities Dohle bodies: They are bluish cytoplasmic inclusions that can be seen in the neutrophils from patients with: • Bacterial infection • Burns • Myelodysplasia • May-Hegglin anomaly • Pregnancy. Alder-Reilly bodies: They are ...
Page 10
... abnormalities in an otherwise healthy appearing child without lymphadenopathy or hepatosplenomegaly almost never is associated with childhood acute leukemia. In such cases, idiopathic thrombocytopenia purpura (ITP) is usually the ...
... abnormalities in an otherwise healthy appearing child without lymphadenopathy or hepatosplenomegaly almost never is associated with childhood acute leukemia. In such cases, idiopathic thrombocytopenia purpura (ITP) is usually the ...
Page 11
... abnormalities (G6PD deficiency) • Asplenia. Fig. 1.3: Target cells (For color version, see Plate 1) TARGET CELLS (FIG. 1.3) Increased surface/volume ratio: • Thalassemia • Hemoglobinopathies — Hb AC or CC — Hb SS, SC, S-Thalassemia ...
... abnormalities (G6PD deficiency) • Asplenia. Fig. 1.3: Target cells (For color version, see Plate 1) TARGET CELLS (FIG. 1.3) Increased surface/volume ratio: • Thalassemia • Hemoglobinopathies — Hb AC or CC — Hb SS, SC, S-Thalassemia ...
Page 19
... abnormality for hemolytic anemia. e. Nail changes: Platynychia, koilonychia, brittle nails are suggestive of iron deficiency. They are less common in children than in adults, but when present are pathognomonic of IDA. Dyskeratotic nails ...
... abnormality for hemolytic anemia. e. Nail changes: Platynychia, koilonychia, brittle nails are suggestive of iron deficiency. They are less common in children than in adults, but when present are pathognomonic of IDA. Dyskeratotic nails ...
Contents
Chapter21 | 156 |
Chapter22 | 165 |
Chapter23 | 178 |
Chapter24 | 181 |
Chapter25 | 199 |
Chapter26 | 208 |
Chapter27 | 232 |
Chapter28 | 247 |
Chapter09 | 69 |
Chapter10 | 75 |
Chapter11 | 85 |
Chapter12 | 90 |
Chapter13 | 97 |
Chapter14 | 115 |
Chapter15 | 120 |
Chapter16 | 125 |
Chapter17 | 130 |
Chapter18 | 144 |
Chapter19 | 148 |
Chapter20 | 153 |
Chapter29 | 259 |
Chapter30 | 262 |
Chapter31 | 266 |
Chapter32 | 272 |
Chapter33 | 281 |
Chapter34 | 289 |
Chapter35 | 298 |
Chapter36 | 305 |
Chapter37 | 309 |
Index | 313 |
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Common terms and phrases
abnormalities acid activity acute adults antibodies anticoagulant antigen aplastic anemia APTT assay associated bleeding blood loss bone marrow transplantation catheter cause chemotherapy child childhood chronic clinical concentration congenital cytopenia decreased defects develop diagnosis disease disorders Disseminated intravascular coagulation donor dose drugs erythrocyte erythropoietin factor VIII ferritin fetal fibrin fibrinogen folate g/dl gene globin granulocytes Haematol hematocrit hematological hemoglobin hemolysis hemolytic anemia hemophilia hemorrhage heparin hepatitis immune increased India infants infection infusion inhibitor intravenous iron deficiency ischemic laboratory leukemia levels liver malignancy mutations myelodysplastic syndrome myeloid neonatal neutropenia newborn normal nutritional occur oral patients Pediatr peripheral plasma platelet platelet count platelet transfusion present preterm prophylaxis protein prothrombin red cell reticulocyte count risk serum severe splenectomy stem cell stroke studies syndrome Table thalassemia therapy thrombin thrombocytopenia thrombocytosis thrombosis tissue treatment usually venous viral virus vitamin