Practical Pediatric HematologyAnupam Sachdeva Evidence-based medicine is the need of the hour. Every pediatrician strives for perfection is his or her practice. These are exciting times for Pediatric Hematology and Oncology. Major advances during the past decade in the field have enhanced the understanding and significantly influenced the management and outcome of many of these chronic and fatal diseases affecting children. While opportunities to learn decrease after one leaves the medical school, science keeps evolving. Keeping this in mind, Indian Academy of Pediatrics (IAP) envisaged starting specialty training under its plan of action 2006. Such trainings keep professional and residents updated about newly launched technologies and other advancements. This book is well illustrated and has many pictures and diagrams for the quick understanding of readers. This will be of use not only to the postgraduates of pediatrics but also will be of immense use to a general pediatrician as a ready-reckoner to be kept on the desk. |
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Results 1-5 of 71
Page 3
... adults and in children between the ages of 2 years and 10 years, the lower limit for MCV is approximately 70 fl + age (in years). The approximate upper limit for MCV is obtained by adding 0.6 fl per year to 84 fl beyond the first year ...
... adults and in children between the ages of 2 years and 10 years, the lower limit for MCV is approximately 70 fl + age (in years). The approximate upper limit for MCV is obtained by adding 0.6 fl per year to 84 fl beyond the first year ...
Page 8
... adult hemoglobin and as the hemoglobin level falls, a threshold of diminishing venous oxygen saturation is reached. A signal in the form of increased erythrocytes creates a physiologic hemoglobin nadir at 7 to 10 weeks of age in normal ...
... adult hemoglobin and as the hemoglobin level falls, a threshold of diminishing venous oxygen saturation is reached. A signal in the form of increased erythrocytes creates a physiologic hemoglobin nadir at 7 to 10 weeks of age in normal ...
Page 19
... adults, but when present are pathognomonic of IDA. Dyskeratotic nails will be seen in dyskeratosis congenita. f. Lymphadenopathy: Significant lymphadenopathy will suggest tuberculosis, HIV, infectious mononucleosis, leukemia, lymphoma ...
... adults, but when present are pathognomonic of IDA. Dyskeratotic nails will be seen in dyskeratosis congenita. f. Lymphadenopathy: Significant lymphadenopathy will suggest tuberculosis, HIV, infectious mononucleosis, leukemia, lymphoma ...
Page 22
... adult due to: 1. Age differences in normal values for Hb/PCV (Table 3.4): Children between the age of 6 months to 12 years appear anemic compared with adults. Their RBCs, however, have a higher concentration of 2,3diphosphoglycerate and ...
... adult due to: 1. Age differences in normal values for Hb/PCV (Table 3.4): Children between the age of 6 months to 12 years appear anemic compared with adults. Their RBCs, however, have a higher concentration of 2,3diphosphoglycerate and ...
Page 23
... adults it is usually secondary to an underlying illness, i.e. a primary cause in the hemopoietic system is more frequent in children. Evaluation of the Anemic Patient Diagnostic approach should begin with a detailed history and physical ...
... adults it is usually secondary to an underlying illness, i.e. a primary cause in the hemopoietic system is more frequent in children. Evaluation of the Anemic Patient Diagnostic approach should begin with a detailed history and physical ...
Contents
Chapter21 | 156 |
Chapter22 | 165 |
Chapter23 | 178 |
Chapter24 | 181 |
Chapter25 | 199 |
Chapter26 | 208 |
Chapter27 | 232 |
Chapter28 | 247 |
Chapter09 | 69 |
Chapter10 | 75 |
Chapter11 | 85 |
Chapter12 | 90 |
Chapter13 | 97 |
Chapter14 | 115 |
Chapter15 | 120 |
Chapter16 | 125 |
Chapter17 | 130 |
Chapter18 | 144 |
Chapter19 | 148 |
Chapter20 | 153 |
Chapter29 | 259 |
Chapter30 | 262 |
Chapter31 | 266 |
Chapter32 | 272 |
Chapter33 | 281 |
Chapter34 | 289 |
Chapter35 | 298 |
Chapter36 | 305 |
Chapter37 | 309 |
Index | 313 |
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abnormalities acid activity acute adults antibodies anticoagulant antigen aplastic anemia APTT assay associated bleeding blood loss bone marrow transplantation catheter cause chemotherapy child childhood chronic clinical concentration congenital cytopenia decreased defects develop diagnosis disease disorders Disseminated intravascular coagulation donor dose drugs erythrocyte erythropoietin factor VIII ferritin fetal fibrin fibrinogen folate g/dl gene globin granulocytes Haematol hematocrit hematological hemoglobin hemolysis hemolytic anemia hemophilia hemorrhage heparin hepatitis immune increased India infants infection infusion inhibitor intravenous iron deficiency ischemic laboratory leukemia levels liver malignancy mutations myelodysplastic syndrome myeloid neonatal neutropenia newborn normal nutritional occur oral patients Pediatr peripheral plasma platelet platelet count platelet transfusion present preterm prophylaxis protein prothrombin red cell reticulocyte count risk serum severe splenectomy stem cell stroke studies syndrome Table thalassemia therapy thrombin thrombocytopenia thrombocytosis thrombosis tissue treatment usually venous viral virus vitamin