Practical Pediatric HematologyAnupam Sachdeva Evidence-based medicine is the need of the hour. Every pediatrician strives for perfection is his or her practice. These are exciting times for Pediatric Hematology and Oncology. Major advances during the past decade in the field have enhanced the understanding and significantly influenced the management and outcome of many of these chronic and fatal diseases affecting children. While opportunities to learn decrease after one leaves the medical school, science keeps evolving. Keeping this in mind, Indian Academy of Pediatrics (IAP) envisaged starting specialty training under its plan of action 2006. Such trainings keep professional and residents updated about newly launched technologies and other advancements. This book is well illustrated and has many pictures and diagrams for the quick understanding of readers. This will be of use not only to the postgraduates of pediatrics but also will be of immense use to a general pediatrician as a ready-reckoner to be kept on the desk. |
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Page 2
... blood thoroughly before analysis.3 Elevated MCH and inaccurate hematocrit ... loss of central pallor in every cell, unlike in true spherocytes, in which ... blood smears. Platelets and Leukocytes Blood smear artifacts similarly can ...
... blood thoroughly before analysis.3 Elevated MCH and inaccurate hematocrit ... loss of central pallor in every cell, unlike in true spherocytes, in which ... blood smears. Platelets and Leukocytes Blood smear artifacts similarly can ...
Page 3
... blood loss in. Basophilic Stippling It is caused by aggregated ribosomes in the erythrocyte (for ribosomal DNA and mitochodrial fragments in lead poisonings) is seen with thalassemia and lead intoxication. Rouleaux Formation It occurs ...
... blood loss in. Basophilic Stippling It is caused by aggregated ribosomes in the erythrocyte (for ribosomal DNA and mitochodrial fragments in lead poisonings) is seen with thalassemia and lead intoxication. Rouleaux Formation It occurs ...
Page 4
... blood loss • Hemolysis. Ancillary measures of erythrocyte destruction include: • Serum bilirubin • Lactate dehydrogenase (LDH). Macrocytosis is caused by an increased number of reticulocytes, which have a large cellular volume (140-150 ...
... blood loss • Hemolysis. Ancillary measures of erythrocyte destruction include: • Serum bilirubin • Lactate dehydrogenase (LDH). Macrocytosis is caused by an increased number of reticulocytes, which have a large cellular volume (140-150 ...
Page 6
... Blood loss • Balanced causes of macrocytic and microcytic anemia, such as iron deficiency combined with folate or B12 deficiency, i.e. dimorphic anemia. However, patients with hemolysis may not be anemic if increased erythropoiesis is ...
... Blood loss • Balanced causes of macrocytic and microcytic anemia, such as iron deficiency combined with folate or B12 deficiency, i.e. dimorphic anemia. However, patients with hemolysis may not be anemic if increased erythropoiesis is ...
Page 7
... Blood loss. The characteristic spur cell in liver disease, which is caused by an alteration in lipid composition of the membrane, is ... blood loss Clinical features Venous pressure Low Laboratory Interpretation of the Complete Blood Count 7.
... Blood loss. The characteristic spur cell in liver disease, which is caused by an alteration in lipid composition of the membrane, is ... blood loss Clinical features Venous pressure Low Laboratory Interpretation of the Complete Blood Count 7.
Contents
Chapter21 | 156 |
Chapter22 | 165 |
Chapter23 | 178 |
Chapter24 | 181 |
Chapter25 | 199 |
Chapter26 | 208 |
Chapter27 | 232 |
Chapter28 | 247 |
Chapter09 | 69 |
Chapter10 | 75 |
Chapter11 | 85 |
Chapter12 | 90 |
Chapter13 | 97 |
Chapter14 | 115 |
Chapter15 | 120 |
Chapter16 | 125 |
Chapter17 | 130 |
Chapter18 | 144 |
Chapter19 | 148 |
Chapter20 | 153 |
Chapter29 | 259 |
Chapter30 | 262 |
Chapter31 | 266 |
Chapter32 | 272 |
Chapter33 | 281 |
Chapter34 | 289 |
Chapter35 | 298 |
Chapter36 | 305 |
Chapter37 | 309 |
Index | 313 |
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Common terms and phrases
abnormalities acid activity acute adults antibodies anticoagulant antigen aplastic anemia APTT assay associated bleeding blood loss bone marrow transplantation catheter cause chemotherapy child childhood chronic clinical concentration congenital cytopenia decreased defects develop diagnosis disease disorders Disseminated intravascular coagulation donor dose drugs erythrocyte erythropoietin factor VIII ferritin fetal fibrin fibrinogen folate g/dl gene globin granulocytes Haematol hematocrit hematological hemoglobin hemolysis hemolytic anemia hemophilia hemorrhage heparin hepatitis immune increased India infants infection infusion inhibitor intravenous iron deficiency ischemic laboratory leukemia levels liver malignancy mutations myelodysplastic syndrome myeloid neonatal neutropenia newborn normal nutritional occur oral patients Pediatr peripheral plasma platelet platelet count platelet transfusion present preterm prophylaxis protein prothrombin red cell reticulocyte count risk serum severe splenectomy stem cell stroke studies syndrome Table thalassemia therapy thrombin thrombocytopenia thrombocytosis thrombosis tissue treatment usually venous viral virus vitamin