Practical Pediatric HematologyAnupam Sachdeva Evidence-based medicine is the need of the hour. Every pediatrician strives for perfection is his or her practice. These are exciting times for Pediatric Hematology and Oncology. Major advances during the past decade in the field have enhanced the understanding and significantly influenced the management and outcome of many of these chronic and fatal diseases affecting children. While opportunities to learn decrease after one leaves the medical school, science keeps evolving. Keeping this in mind, Indian Academy of Pediatrics (IAP) envisaged starting specialty training under its plan of action 2006. Such trainings keep professional and residents updated about newly launched technologies and other advancements. This book is well illustrated and has many pictures and diagrams for the quick understanding of readers. This will be of use not only to the postgraduates of pediatrics but also will be of immense use to a general pediatrician as a ready-reckoner to be kept on the desk. |
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Page xxii
... Hepatitis B Virus 199 Hepatitis C Virus 201 Hepatitis D Virus 202 Hepatitis A Virus 202 Hepatitis E Virus 202 Hepatitis GB Virus C/Hepatitis G Virus TT Virus 202 Human Immunodeficiency Virus Types 1 and 2 (HIV1 and HIV2) 202 Human T ...
... Hepatitis B Virus 199 Hepatitis C Virus 201 Hepatitis D Virus 202 Hepatitis A Virus 202 Hepatitis E Virus 202 Hepatitis GB Virus C/Hepatitis G Virus TT Virus 202 Human Immunodeficiency Virus Types 1 and 2 (HIV1 and HIV2) 202 Human T ...
Page 10
... Hepatitis A and B • Respiratory syncytial virus • Rubella. Changes usually are noted within 1 to 2 days of infection and often can persist for several weeks. If the clinical situation warrants, consideration should be given to ...
... Hepatitis A and B • Respiratory syncytial virus • Rubella. Changes usually are noted within 1 to 2 days of infection and often can persist for several weeks. If the clinical situation warrants, consideration should be given to ...
Page 11
... hepatitis. White blood cell counts of more than 30,000/mm3 with 60 to 70% lymphocytes, especially if they are described as clefted or baby bottom may be caused by pertusis. Lymphopenia is associated with corticosteroid therapy ...
... hepatitis. White blood cell counts of more than 30,000/mm3 with 60 to 70% lymphocytes, especially if they are described as clefted or baby bottom may be caused by pertusis. Lymphopenia is associated with corticosteroid therapy ...
Page 18
... hepatitis virus. G6PD deficiency induced hemolysis can be precipitated by many infections and drugs used to treat such infections. Hemolysis could also be induced by malaria. Bone marrow suppression can occur following many viral ...
... hepatitis virus. G6PD deficiency induced hemolysis can be precipitated by many infections and drugs used to treat such infections. Hemolysis could also be induced by malaria. Bone marrow suppression can occur following many viral ...
Page 57
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Contents
Chapter21 | 156 |
Chapter22 | 165 |
Chapter23 | 178 |
Chapter24 | 181 |
Chapter25 | 199 |
Chapter26 | 208 |
Chapter27 | 232 |
Chapter28 | 247 |
Chapter09 | 69 |
Chapter10 | 75 |
Chapter11 | 85 |
Chapter12 | 90 |
Chapter13 | 97 |
Chapter14 | 115 |
Chapter15 | 120 |
Chapter16 | 125 |
Chapter17 | 130 |
Chapter18 | 144 |
Chapter19 | 148 |
Chapter20 | 153 |
Chapter29 | 259 |
Chapter30 | 262 |
Chapter31 | 266 |
Chapter32 | 272 |
Chapter33 | 281 |
Chapter34 | 289 |
Chapter35 | 298 |
Chapter36 | 305 |
Chapter37 | 309 |
Index | 313 |
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Common terms and phrases
abnormalities acid activity acute adults antibodies anticoagulant antigen aplastic anemia APTT assay associated bleeding blood loss bone marrow transplantation catheter cause chemotherapy child childhood chronic clinical concentration congenital cytopenia decreased defects develop diagnosis disease disorders Disseminated intravascular coagulation donor dose drugs erythrocyte erythropoietin factor VIII ferritin fetal fibrin fibrinogen folate g/dl gene globin granulocytes Haematol hematocrit hematological hemoglobin hemolysis hemolytic anemia hemophilia hemorrhage heparin hepatitis immune increased India infants infection infusion inhibitor intravenous iron deficiency ischemic laboratory leukemia levels liver malignancy mutations myelodysplastic syndrome myeloid neonatal neutropenia newborn normal nutritional occur oral patients Pediatr peripheral plasma platelet platelet count platelet transfusion present preterm prophylaxis protein prothrombin red cell reticulocyte count risk serum severe splenectomy stem cell stroke studies syndrome Table thalassemia therapy thrombin thrombocytopenia thrombocytosis thrombosis tissue treatment usually venous viral virus vitamin