Practical Pediatric HematologyAnupam Sachdeva Evidence-based medicine is the need of the hour. Every pediatrician strives for perfection is his or her practice. These are exciting times for Pediatric Hematology and Oncology. Major advances during the past decade in the field have enhanced the understanding and significantly influenced the management and outcome of many of these chronic and fatal diseases affecting children. While opportunities to learn decrease after one leaves the medical school, science keeps evolving. Keeping this in mind, Indian Academy of Pediatrics (IAP) envisaged starting specialty training under its plan of action 2006. Such trainings keep professional and residents updated about newly launched technologies and other advancements. This book is well illustrated and has many pictures and diagrams for the quick understanding of readers. This will be of use not only to the postgraduates of pediatrics but also will be of immense use to a general pediatrician as a ready-reckoner to be kept on the desk. |
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Results 1-5 of 85
Page 2
... Liver disease 3. Hemoglobinopathies (hemoglobins C, D, and E) 4. Thalassemia 5. Postsplenectomy state 6. Hereditary xerocytosis 7. Lecithin cholesterol acyl transferase deficiency (LCAT deficiency). Howell-Jolly Bodies They are nuclear ...
... Liver disease 3. Hemoglobinopathies (hemoglobins C, D, and E) 4. Thalassemia 5. Postsplenectomy state 6. Hereditary xerocytosis 7. Lecithin cholesterol acyl transferase deficiency (LCAT deficiency). Howell-Jolly Bodies They are nuclear ...
Page 6
... Liver disease. Anemia of renal disease is due to: • Erythropoietin insufficiency18 • Serum inhibitors of erythropoiesis may accumulate in the uremic patients • Hyperparathyroidism may inhibit erythropoiesis by promoting myelofibrosis.19 ...
... Liver disease. Anemia of renal disease is due to: • Erythropoietin insufficiency18 • Serum inhibitors of erythropoiesis may accumulate in the uremic patients • Hyperparathyroidism may inhibit erythropoiesis by promoting myelofibrosis.19 ...
Page 7
... liver disease is multifactorial and includes: • Hypersplenism • Concomitant vitamin-nutritional deficiency • Blood loss. The characteristic spur cell in liver disease, which is caused by an alteration in lipid composition of the ...
... liver disease is multifactorial and includes: • Hypersplenism • Concomitant vitamin-nutritional deficiency • Blood loss. The characteristic spur cell in liver disease, which is caused by an alteration in lipid composition of the ...
Page 11
... liver disease • Postsplenectomy or hyposplenic states • Severe iron deficiency • Hb E (heterozygote and homozygote) • LCAT deficiency • Abetalipoproteinemia. SPHEROCYTES (FIG. 1.4) Decreased surface/volume ratio, hyperdense (>MCHC ...
... liver disease • Postsplenectomy or hyposplenic states • Severe iron deficiency • Hb E (heterozygote and homozygote) • LCAT deficiency • Abetalipoproteinemia. SPHEROCYTES (FIG. 1.4) Decreased surface/volume ratio, hyperdense (>MCHC ...
Page 12
... Liver disease • Pyruvate kinase deficiency • Peptic ulcer disease or gastric carcinoma • After blood transfusion. SCHISTOCYTES (FIG. 1.7) • Malignant hypertension • Systemic amyloidosis • Liver cirrhosis • Disseminated carcinomatosis ...
... Liver disease • Pyruvate kinase deficiency • Peptic ulcer disease or gastric carcinoma • After blood transfusion. SCHISTOCYTES (FIG. 1.7) • Malignant hypertension • Systemic amyloidosis • Liver cirrhosis • Disseminated carcinomatosis ...
Contents
Chapter21 | 156 |
Chapter22 | 165 |
Chapter23 | 178 |
Chapter24 | 181 |
Chapter25 | 199 |
Chapter26 | 208 |
Chapter27 | 232 |
Chapter28 | 247 |
Chapter09 | 69 |
Chapter10 | 75 |
Chapter11 | 85 |
Chapter12 | 90 |
Chapter13 | 97 |
Chapter14 | 115 |
Chapter15 | 120 |
Chapter16 | 125 |
Chapter17 | 130 |
Chapter18 | 144 |
Chapter19 | 148 |
Chapter20 | 153 |
Chapter29 | 259 |
Chapter30 | 262 |
Chapter31 | 266 |
Chapter32 | 272 |
Chapter33 | 281 |
Chapter34 | 289 |
Chapter35 | 298 |
Chapter36 | 305 |
Chapter37 | 309 |
Index | 313 |
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Common terms and phrases
abnormalities acid activity acute adults antibodies anticoagulant antigen aplastic anemia APTT assay associated bleeding blood loss bone marrow transplantation catheter cause chemotherapy child childhood chronic clinical concentration congenital cytopenia decreased defects develop diagnosis disease disorders Disseminated intravascular coagulation donor dose drugs erythrocyte erythropoietin factor VIII ferritin fetal fibrin fibrinogen folate g/dl gene globin granulocytes Haematol hematocrit hematological hemoglobin hemolysis hemolytic anemia hemophilia hemorrhage heparin hepatitis immune increased India infants infection infusion inhibitor intravenous iron deficiency ischemic laboratory leukemia levels liver malignancy mutations myelodysplastic syndrome myeloid neonatal neutropenia newborn normal nutritional occur oral patients Pediatr peripheral plasma platelet platelet count platelet transfusion present preterm prophylaxis protein prothrombin red cell reticulocyte count risk serum severe splenectomy stem cell stroke studies syndrome Table thalassemia therapy thrombin thrombocytopenia thrombocytosis thrombosis tissue treatment usually venous viral virus vitamin